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Although it has been commented that wind tunnel testing showed that NASA's design was already ideal,[16] the shape requirements were mandated by its potential military capabilities to transport large payloads to low Earth orbit, themselves a counterpart to the Pentagon's initially projected missions for the Shuttle.[17] Even though the Molniya Scientific Production Association proposed its Spiral programme design[18] (halted 13 years earlier), it was rejected as being altogether dissimilar from the American shuttle design. At the beginning of the 1970s, the development work on the Soviet space program Buran took concrete shape, the construction of the first space shuttle finally began in 1980. On November 1, 1988, the Buran 1.01 started on its first and ultimately only unmanned space flight. In addition to the external similarity to the NASA space shuttle, there were other similarities, such as the concept of starting with a powerful launch vehicle, or the transportation of the shuttle within the earth's atmosphere between orbital flights. While the Americans converted two Boeing 747s that were already getting on in years, the Soviets decided to develop a new carrier aircraft based on the Antonov AN-124, which was equipped with six turbofans to ensure the appropriate performance and payload. it succeeded and on December 21, 1988, the largest aircraft of all time took off on its maiden flight. The payload of an incredible 34 tons, 110 tons of which was outboard, was enough to transport the Buran piggyback to the Kazakh cosmodrome Baikonur or to the Paris Air Show in 1989. The authorities of Kazakhstan are considering the possibility of using the second flight model of the Soviet space shuttle Buran in their museum expositions dedicated to the history of the development of the Baikonur cosmodrome. This was reported to TASS on Thursday by the press service of the Ministry of Digital Development, Innovation and Aerospace Industry of Kazakhstan. On November 17, Dmitry Rogozin, general director of the state corporation Roscosmos, told reporters that the second flight model of the Soviet space shuttle Buran, its mock-up and the Energia rocket could be transferred to a state or private museum if Roskosmos finds their owner and negotiates with them. Later, Dauren Musa, CEO of the Baikonur Rocket and Space Company Kazakhstan Joint-Stock Company, announced in his microblog on Twitter that his company owns Buran , and also its layout. At the same time, the press service of the Ministry of Digital Development, Innovation and Aerospace Industry of Kazakhstan reported that the authorities of the republic plan to return the second flight model of the Soviet space shuttle Buran to state property. In filed a claim for liquidation of the spacecraft "Buran". The next meeting is scheduled in the Specialized Interdistrict Economic Court of the city of Alma-Ata on December 10 of this year. The most powerful Soviet booster rocket, first launched on 15 May 1987. Used to launch the Soviet space shuttle Buran in 1968, the SL-17 Energiya booster was capable, with the aid of strap-on boosters, of launching payloads of up to 190 tonnes into Earth orbit. 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Have not tried the one at Carolina place or down at South Park but geez who are you trying to Impress? My daughter-in-law went to the one in Monroe and she told me she will not go back and that is exactly how I feel . That wonderley clothing is horrific! You have lost 2 devoted customers due to terrible merchandise buying on a buyers account! went to belk in Laurinburg, North Carolina 28352 and saw a pair of shoes I wanted on the discount shelf. I took the shoe to the scanner and it said the shoes was $90.00 and $89.99 discount, my cost $.01 cent. went to checkout and they rain up $.01 and the cashier said she could not give them to me for that even those it rain up for that price. My thought was if that was the price rain up that is what I supose to pay. she gave me the shoes for a good price $13.19, but I still was not happy. shoe #misc mdse 99999999. I love belk, but that make me more careful when I am shopping in belk about how they can change pricing whenever they want. 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You can find here Belk mailing and postal address, corporate office address, customer service help number, corporate headquarters address of Belk, Customer service support phone number, corporate office HQ Address. William Belk's success resulted from some retailing ideas that were innovative for the time. In 1897, he combined the purchasing power of the four stores, plus two others in which the brothers had no financial interest, and formed a loose cooperative buying network. This allowed them to purchase goods in bulk quantity at favorable prices. All purchases and sales were cash. Belk also made extensive use of advertising. In 1899, the brothers opened a store in Greensboro, North Carolina. The Belks' private-label business was now thriving, and by 1959 it accounted for a major share of the buying office's inventory. In the late 1950s, the Belks department stores had nearly peaked in the South, with 325 stores in 16 states. In 1956, Belk acquired its only viable competitor in the region, the Efird department stores. Under the direction of president and CEO Thomas Belk, the company succeeded in transforming its operations in order to meet the demands of style-conscious shoppers during the 1980s, as opposed to catering strictly to a clientele looking for thrift, durability, and value. For example, Belk hosted its first fashion buying show in New York in 1983, and within four years representatives from the nation's top fashion lines were competing for representation in the show. This further consolidated buying and proved Belk's place in the fashion retail market. Marking the change from bargain chain to fashion stores was top designer Oscar de la Renta's appearance at a grand reopening of a Belk store in 1986. Though some stores retained the small-town, bargain-budget flavor of Belk's founder's vision, others moved to suburban malls and shopping centers. The company celebrated its 100th birthday in 1988 while opening a huge new BSS office complex in Charlotte. The company would later close its New York office and consolidate buying operations at the new facility. Belk was expanding, adding plenty of retail space on the home front and in new territories. In 2002, the company added 40,000 square feet to its store at Charlotte's SouthPark Mall and opened a 180,000-square-foot location near Durham's Research Triangle Park. (Another 180,000-square-foot store had opened in Charleston two years earlier.) Belk consolidated its merchandising, formerly handled by the four divisions, into one central buying office during the year. John M. Belk, son of the company's founder, retired in May 2004. He was succeeded as chairman and CEO by Thomas M. "Tim" Belk, Jr., who had previously been Belk, Inc.'s president of store divisions, human resources, real estate, and visual presentation. Tim Belk's brothers H.W. McKay and John R. Belk were appointed chief merchandising officer and chief operating officer, respectively, and remained co-presidents with Tim Belk. Belk was founded by William Henry Belk in 1888 in Monroe, North Carolina. The store was originally called New York Racket and then renamed to Belk Brothers.In 1908, the company headquarters was moved to Charlotte, where it remains today. A huge flagship store was built where the Bank of America Corporate Center now stands.if(typeof ez_ad_units!='undefined'){ez_ad_units.push([[300,250],'corporateofficehq_com-box-4','ezslot_2',174,'0','0'])};__ez_fad_position('div-gpt-ad-corporateofficehq_com-box-4-0'); In 1956, department store chain Efird is acquired.In 1988, a new corporate headquarters is constructed in Charlotte.In 1998, the company consolidates its 112 brands to form Belk Inc.In 2004, John M. Belk retires as CEO after 50 years of leading the company. He is replaced by Thomas Belk.In April of 2015, Belk was sold to a private equity firm, Sycamore Partners. Since then, it has laid off more than 200 persons from the corporate office and slashed jobs across the board at all 300 store locations.In June 2018, the company was listed as one of the three worst employers to work for in North Carolina.Today, there are over 300 Belk department stores in the US.Headquarters remain in Charlotte, North Carolina, at this time. "Times change. How people are buying is changing. We have a new generation of Millenials who probably have never heard of Belk or certainly don't know the history. They are now the dominant buyers in the retail area," said N.C. State University economics professor Mike Walden. I would like to tell you about someone that checked me out recently. The first lady could not get my rewards and belk gift card to go through. After many tries she called over another person. They both tried everything to get it right. Finally they called someone else. She could not get the register to work properly and moved me to her register in another section. It turned out that all the registers in the store seemed to be causing problems. This lady was very apologetic and worked hard to complete my purchase. She called the help desk to no avail because there were so many people ahead of her. She finally managed to complete my purchase. Her name is Sharon and she is at store 843-797-2600. You couldn't ask for a nicer and helpful employee. Your store has been one of a few southern dept stores in Texas that I enjoyed shopping andpurchasing from, but hearing that you have changed your "social Stance" whatever that meansand will not let the Salvation army bell ringers in front of your store upsets me greatly.I hope someone in your corporate office will change this before its too late, southern peoplewill shop elsewhere and that includes me. I expected more from your company. 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LabCorp, based out of Burlington, operates a sophisticated laboratory network with more than 34,000 employees and 220 thousand clients, including physician offices, hospitals, managed care organizations and biotechnology and pharmaceutical companies. He held key top management positions in the company's central buying office organization and became president of the various Belk Inc. store corporations in the early 1950s. Mr. Belk served as the company's chief executive until his retirement in May 2004. "He will be truly missed," Williams said. Our platform is designed to provide you with a worry-free John M. Belk Arena ticket buying experience. We use the highest standards in the online ticket exchange industry to ensure your safety when you are making your purchase. TicketSmarter has a 100% guarantee that you will receive valid tickets for your event just as if you had made your purchase from the ticket office at John M. 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Glean insights from case-studies of real-world applications of AI-based tools: learn about clinical trials/drug development basics, fundamental principles of AI, ML and DL, opportunities and challenges to development and adoption (including data sharing and privacy, device standardization, regulatory aspects, and transparency). Abstract Quality of clinical trials has improved steadily over last two decades, but certain areas in trial methodology still require special attention like in sample size calculation. The sample size is one of the basic steps in planning any clinical trial and any negligence in its calculation may lead to rejection of true findings and false results may get approval. Although statisticians play a major role in sample size estimation basic knowledge regarding sample size calculation is very sparse among most of the anesthesiologists related to research including under trainee doctors. In this review, we will discuss how important sample size calculation is for research studies and the effects of underestimation or overestimation of sample size on project's results. We have highlighted the basic concepts regarding various parameters needed to calculate the sample size along with examples. The following shows the number of patients that take part and the purpose of the most common phases. Although the trial phases are explained in the context of drug treatment trials, the same concepts apply to most types of clinical trials. Fundamentals of Clinical and Translational Science (FunCaTS) is a combination of 13 online modules strategically packaged together to enable medical professionals and allied health staff to expand their knowledge of the components of clinical and translational research. These components provide the fundamental knowledge to promote understanding of the process of bringing discoveries to the bedside and then to the population. To maximize this costly investment, pharmaceutical companies have a strong interest in testing new products in multiple markets worldwide simultaneously. Although some of the key concepts and rules governing the conduct of clinical trials and research are to a certain extent harmonized in some parts of the world, important differences are still manifold. SUMMER COURSES IN EPIDEMIOLOGY, 1997The Johns Hopkins University School of Hygiene and Public Health is sponsoring the Fifteenth Annual Graduate Summer Program in Epidemiology, to be conducted from 16 June to 4 July 1997. The program includes: principles of epidemiology; introduction to biostatistics; methods in epidemiology; intermediate biostatistics; applications of the case-control method; epidemiologic methods for planning and evaluating health services; epidemiologic basis for tuberculosis control; design and conduct of clinical trials; cohort studies; methods of health risk assessment; infectious disease epidemiology; nutritional epidemiology; outcomes and effectiveness research; epidemiology of AIDS; molecular biology: a primer for epidemiologists; epidemiology of emerging infections; use of microcomputers in epidemiology; advanced topics in SAS programming; meta-analysis; epidemiology and public policy; statistical methods for longitudinal data; new paradigms/new approaches to management of epidemiological studies. Proficiency in the English language is required.Further information is available from: Helen Walters, Program Coordinator, Graduate Summer Program in Epidemiology, The Johns Hopkins University, School of Hygiene and Public Health, 615 North Wolfe Street, Baltimore, Maryland 21205. Tel. (410) 955-7158; Fax(410) 955-0863; E-mail: HWALTERS@PHNET.SPH.JHU.EDUThe University of Michigan School of Public Health announces the Thirtieth-two International Graduate Summer Session in Epidemiology to be conducted from 6 to 25 July, 1997. Three, two and one-week courses will be offered. Three-week courses include: applied epidemiology for health practitioners; fundamentals of epidemiology; and fundamentals of biostatistics. Two-week courses include: basic computer applications in epidemiology; genetic epidemiology; and Methods in Medical Quality Assessment.One-week courses include: basic concepts of clinical epidemiology; clinical trials, design and conducts; clinical trials-analytic methods; introduction to the logistic model; analysis of survival, and follow-up data; introduction to cancer epidemiology; advanced concepts and methods in cancer epidemiology; cancer prevention; occupational epidemiology; enviromental epidemiology and risk assesment; epidemiology of injuries; epidemiologic methods and injury control; epidemiology of mental disorders; update in infectious diseases; emerging infections; sexually transmitted diseases and HIV; nutritional epidemiology; epidemiologic issues in women's health: controversies and challenges; pharmacoepidemiolgy; scientific writing; advanced computer applications in epidemiology; behavioral modification; ethics, epidemiology and public healthresearch; introduction to genetics in Public Health; epidemiology and the law; cardiovascular epidemiology; and Meta-Analysis: A synthesis of Medical Literature. Proficiency in the English Language is needed.For further information contact: Dr. David Schottenfeld or JodyGray, Administrative Coordinator, Graduate Summer Session in Epidemiology, The University of Michigan, School of Public Health, 109 S. Observatory Street, Ann Arbor, Michigan 48109-2029. Tel. (313) 764-5454; Fax (313) 764-3192; E-mail: umichgss@sph.umich.eduThe Department of Epidemiology and Biostatistics, McGill Universitywill hold its Twelveth Annual Summer Program in Epidemiology and Biostatistics from 5 May to 27 June, 1997. General topics willinclude: principles and methods of epidemiology; statistical inference; clinical epidemiology; multivariate analysis; practical issues in protocol development. For more information contact: Summer Program Coordinator, Joint Departments of Epidemiology, Biostatistics and Occupational Health, McGill University, 1020 Pine Avenue West, Room 38B, Montreal, Quebec, Canada, H3A 1A2. Tel. (514) 398-3973; Fax (514) 398-4503; E-mail: Therefore, to explore the need for a new instrument or a modifiable instrument, an initial review of qualitative literature [7,8,9,10,11,12,13,14, 16, 17, 26,27,28], the transcript of a 2018 FDA advisory committee meeting discussing drug development for the treatment of children with achondroplasia [15], clinical expert interviews, and existing COA measures was conducted to gain an initial understanding of the lived experience of achondroplasia and review potentially relevant COAs. Two measures, the Childhood Health Assessment Questionnaire (CHAQ) [29, 30] and Quality of Life in Short Stature Youth (QoLISSY) Brief [31] were selected for further investigation in this interview study based on their conceptual coverage, with a focus on assessment of physical functioning, pain and emotional/social well-being, respectively. Additionally, patient-reported and observer-reported outcome (ObsRO) versions of the CHAQ and QoLISSY-Brief were available which could allow some children/adolescents to self-report on their own condition. While caregiver report is necessary and appropriate for children who are too young or otherwise unable to report for themselves, patient-report for older children/adolescents should not be overlooked as the affected individual is most accurately able to report their experiences, particularly for non-observable concepts such as feelings and emotions [32]. This free one-day event brings together clinical cancer researchers and biostatisticians to discuss new and fundamental concepts in the design and implementation of therapeutic and non-therapeutic interventional trials. TRACTION scientists execute patient-driven translational research to advance a portfolio of novel therapeutic concepts into clinical trials. As a result of deep integration with MD Anderson investigators and the support of the Moon Shots Program and its enabling platforms, TRACTION is making it possible to advance the most innovative therapies into clinical proof of concept studies in defined patient populations. The curriculum of the Clinical Trials Management Online Certificate covers core concepts consistent with the Association of Clinical Research Professionals (ACRP) certification exams, a valuable credentialing opportunity for clinical research professionals. For exam details, including eligibility requirements, visit Association of Clinical Research Professionals. It is expected that students pursuing the Clinical Trials Management Certificate will have a strong science and/or health-care related background. In addition, two years of college level coursework is recommended. The certificate provides the fundamental knowledge to enter the field of clinical research. While the estimand describes the target of estimation, in practice, teams designing clinical trials are unlikely to come up with a clear and complete objective without discussing the estimand attributes and, in particular, potential intercurrent events. Hence, defining study objectives should be an iterative process, being fine-tuned using the estimand framework as a tool to facilitate such discussion. The clear trial objective would then directly imply how key intercurrent events will be handled and what is the treatment effect of interest. The goal of clarifying the clinical research question more precisely is not new, however, and other concepts such as the PICO model have been previously proposed [3]. The estimand framework differs from the PICO model as it highlights the importance of intercurrent events ensuring that summary measures used to compare treatment conditions are made explicit. It may also in the future potential lead to increased standardisation. The lack of standardized analysis and interpretation of PRO data has been recognized by a number of focus groups working with PRO data in various areas. Within oncology, for example the lack of standardisation is being addressed by a multi-stakeholder, international consortium, SISAQOL [4]. The estimand framework provides common language which can be applied to the initial SISAQOL recommendations, which have focused on developing a taxonomy of research objectives that can be matched with appropriate statistical [5] Extending these to describe more fully within the estimand framework will provide a clear direction for implementation of these recommendations. 59ce067264 https://www.powerandpoise.org/group/mysite-231-group/discussion/180ef828-3bdd-4634-b995-aaef6e3d23db

Matrix Hearts V1.2 Click Here >> https://urllie.com/2tlMXG Abstract:Single-cell Assay for Transposase-Accessible Chromatin using sequencing (scATAC-seq) is rapidly becoming a powerful technology for assessing the epigenetic landscape of thousands of cells. However, the sparsity of the resulting data poses significant challenges to their interpretability and informativeness. Different computational methods are available, proposing ways to generate significant features from accessibility data and process them to obtain meaningful results. Foremost among them is the peak calling, which interprets the raw scATAC-seq data generating the peaks as features. However, scATAC-seq data are not trivially comparable with single-cell RNA sequencing (scRNA-seq) data, an increasingly pressing challenge since the necessity of multimodal experiments integration. For this reason, this study wants to improve the concept of the Gene Activity Matrix (GAM), which links the accessibility data to the genes, by proposing an improved version of the Genomic-Annotated Gene Activity Matrix (GAGAM) concept. Specifically, this paper presents GAGAM v1.2, a new and better version of GAGAM v1.0. GAGAM aims to label the peaks and link them to the genes through functional annotation of the whole genome. Using genes as features in scATAC-seq datasets makes different datasets comparable and allows linking gene accessibility and expression. This link is crucial for gene regulation understanding and fundamental for the increasing impact of multi-omics data. Results confirm that our method performs better than the previous GAMs and shows a preliminary comparison with scRNA-seq data.Keywords: epigenomic single-cell data; gene activity matrix; bioinformatics [Added in v1.2]restrictedThis room can be joined if you were invited or if you are a member of anotherroom listed in the join rules. If the server cannot verify membership for anyof the listed rooms then you can only join with an invite. Note that this ruleis only expected to work in room versions which support it. Some message types support HTML in the event content that clients shouldprefer to display if available. Currently m.text, m.emote, andm.notice support an additional format parameter oforg.matrix.custom.html. When this field is present, a formatted_bodywith the HTML must be provided. The plain text version of the HTMLshould be provided in the body. Clients that do support rich replies MUST provide the fallback format onreplies, and MUST strip the fallback before rendering the reply. Richreplies MUST have a format of org.matrix.custom.html and therefore aformatted_body alongside the body and appropriate msgtype. Thespecific fallback text is different for each msgtype, however thegeneral format for the body is: To send spoilers clients MUST use the formatted_body and therefore theorg.matrix.custom.html format, described above. This makes spoilers valid onany msgtype which can support this format appropriately. Our understanding of the spatiotemporal regulation of cardiogenesis is hindered by the difficulties in modeling this complex organ currently by in vitro models. Here we develop a method to generate heart organoids from mouse embryonic stem cell-derived embryoid bodies. Consecutive morphological changes proceed in a self-organizing manner in the presence of the laminin-entactin (LN/ET) complex and fibroblast growth factor 4 (FGF4), and the resulting in vitro heart organoid possesses atrium- and ventricle-like parts containing cardiac muscle, conducting tissues, smooth muscle and endothelial cells that exhibited myocardial contraction and action potentials. The heart organoids exhibit ultrastructural, histochemical and gene expression characteristics of considerable similarity to those of developmental hearts in vivo. Our results demonstrate that this method not only provides a biomimetic model of the developing heart-like structure with simplified differentiation protocol, but also represents a promising research tool with a broad range of applications, including drug testing. For 3D culture methods using ESCs or adult stem cells, ECMs including Matrigel and laminin were used for in vitro organogenesis, including the development of the brain16, stomach17, kidney18, and intestinal tissues19. FGF and FGFR1 are required for cardiac gene expression, particularly during SHF proliferation and chamber formation in cooperation with the Sonic hedgehog gene during heart development20,21. Therefore, it may be beneficial to consider the role of the extracellular matrix (ECM) environment and FGF signal, which may promote the self-organizing process reconstructing in vivo cardiogenesis. The mouse model system is effective because the embryonic period of mice is shorter than that of humans and advantageous because embryonic heart samples are easily obtained for comparison, and many ESC lines with mutations in genes related to heart development are available for comprehensive analysis. Here, we report the generation of a heart organoid with atrium- and ventricle-like structures showing the ultrastructural, histochemical and gene expression profile similarities to embryonic hearts in the presence of the laminin-entactin (LN/ET) complex22 in the ECM and exogenous fibroblast growth factor 4 (FGF4)20 via consecutive morphological changes like self-organization and, therefore, without the requirement of complex differentiation protocols. Next, we investigated whether in vitro heart organoids faithfully exhibit cardiac properties. The ultrastructural and structural compartments of heart organoids, as well as their development, were evaluated through comparison with embryonic mouse hearts. Transmission electron microscopy revealed that the heart organoids exhibited the CM-like ultrastructures that appear during embryogenesis, including sarcomere structures with Z-bands, mitochondria, and desmosomes (Fig. 2a). Additionally, further distinctive characteristics, such as the honeycomb structure of the sarcoplasmic reticulum (SR) (Fig. 2b, left) and intercalated disc (ID, and especially wavy ventricular myocyte IDs; Fig. 2b, right)4, were observed in the heart organoids. The ID is a cardiac muscle-specific structure required for coordinated muscle contraction, indicating that these heart organoids might possess contractile cardiac muscle cell properties. To confirm whether the heart organoids (Fig. 2c, left) expressed cardiac transcription factors (TFs) and/or structural genes expressed by the in vivo embryonic heart, we assessed the expression of T-box transcriptional factor 5 (Tbx5, an important marker for proper development of both atria and the LV27,28,29,30,31) and SM-myosin heavy chain (SM-MHC, an SM marker). Notably, the spatial pattern of Tbx5 expression in the heart organoids resembled that of in vivo embryonic hearts (E12.5H and E13.5H) after chamber formation (i.e., positive in the left atrium-, right atrium- and left ventricle-like parts) and also detected SM-MHC expression (Fig. 2c, right). Taken together, these results suggested that FGF4 and the gelated LN/ET complex are required for effective in vitro generation of heart organoids with similar cardiac properties to those of embryonic hearts. In the cardiogenesis, several types of cardiac cells and neuronal elements are differentiated from undifferentiated cells. To assess whether undifferentiated stem cells remained in the heart organoids cultured for 11 days, we performed immunostaining assays to detect the pluripotent stem cell marker Oct3/4. Concerning undifferentiated stem cell markers the heart organoids did not exhibit any Oct3/4 expression, in contrast to the EBs prior to culture, indicating that no pluripotent stem cells remained in the 11-day cultured heart organoid (Supplementary Fig. 5a), whereas Nestin which is expressed in the fetal heart as well as in brain tissue was detected in both of the heart organoids and E12.5 hearts. Based on these results, the heart organoids appeared to exhibit the organized structural properties of the embryonic hearts (CMs, SMs and ECs) as well as the cytological and histological maturity, such as the presence of Purkinje fibers, the association of SR/T tubule junction and the tight neuronal contact with the myocytes. Next, we investigated the expressions of coronary circulation-related genes in the heart organoids. The development of coronary circulation is heralded by the appearance of budding and canalized venous sprouts from the sinus venosus which contains vein cells undergo an early cell fate switch to create pre-artery population to build coronary arteries4. Especially, Nr2f2 (Coup-tf2), a venous transcription factor, regulates the expression of artery genes. Notably, the gene expression level of the venous marker Nr2f2 in the heart organoids was similar to that in in vivo embryonic hearts (Supplementary Fig. 5c, top). Similarly, in almost all of the heart organoids (regardless of the culture period), the gene expression patterns of the artery markers Cxcr4, Jag2 and Msx1 were similar to those of in vivo embryonic hearts from E9.5 to E12.5, although other artery markers, including Notch4, Dll4 and Gja4, were only expressed at low levels in heart organoids cultured for 6 days (Supplementary Fig. 5c, bottom). These results indicated that, overall, the heart organoid mimics the specific features of in vivo embryonic hearts in gene expressions, reflecting the diverse property of mammalian cardiac development including coronary circulation. Also, the expression of Nestin (fetal heart and brain marker) was observed in both heart organoids and in E12.5 hearts, suggesting that Nestin-positive cells derived from NCCs may contribute to cardiac innervation and conduction through the cardiac autonomic nervous system25,53. Despite major improvements in the treatment of virtually all cardiac disorders, heart failure (HF) is an exception, in that its prevalence is rising, and only small prolongations in survival are occurring. An increasing fraction, especially older women with diabetes, obesity, and atrial fibrillation exhibit HF with preserved systolic function. Several pathogenetic mechanisms appear to be operative in HF. These include increased hemodynamic overload, ischemia-related dysfunction, ventricular remodeling, excessive neurohumoral stimulation, abnormal myocyte calcium cycling, excessive or inadequate proliferation of the extracellular matrix, accelerated apoptosis, and genetic mutations. Biomarkers released as a consequence of myocardial stretch, imbalance between formation and breakdown of extracellular matrix, inflammation, and renal failure are useful in the identification of the pathogenetic mechanism and, when used in combination, may become helpful in estimating prognosis and selecting appropriate therapy. Promising new therapies that are now undergoing intensive investigation include an angiotensin receptor neprilysin inhibitor, a naturally-occurring vasodilator peptide, a myofilament sensitizer and several drugs that enhance Ca++ uptake by the sarcoplasmic reticulum. Cell therapy, using autologous bone marrow and cardiac progenitor cells, appears to be promising, as does gene therapy. Chronic left ventricular assistance with continuous flow pumps is being applied more frequently and successfully as destination therapy, as a bridge to transplantation, and even as a bridge to recovery and explantation. While many of these therapies will improve the care of patients with HF, significant reductions in prevalence will require vigorous, multifaceted, preventive approaches. 59ce067264 https://www.brdsrvs.com/group/mysite-200-group/discussion/ea349231-cb56-400d-8d23-2648b4937324

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You can typically detach these filters by unscrewing them and lifting them out of the vacuum. Some models may have two filters that both need to be removed. The Pure Cool was different from other Dyson products in a key way, though: at the time, it was the company's only smart, internet-connected device (the Dyson Eye robot vacuum soon followed). Since then, Dyson has made clear its desire to integrate connectivity and AI into more of its products and become a major smart-home player, opening up a huge research facility in Singapore last year to invest in a future where \"everything is automatic.\" A few Dyson reps dropped by the Mashable offices in New York City to walk me through what's new with the Pure Cool. 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Dyson says if you run the purifier for 12 hours a day, the filters should last about a year (a little over 4,000 hours total), though many households -- especially those that are empty during the day -- likely won't need to run it that much. Filter replacements cost $79.99 ($10 more than the previous model). HEPA filters remove fine particles like smoke or pollen and mold fragments from the air, but like any filter they are eventually full of debris. As the filter fibers get clogged, the purifier will lose airflow and fewer particles will be captured. With HEPA filters being used in vacuum cleaners, air purifiers and HVAC systems, you may have wondered how to clean a HEPA filter instead of replacing it numerous times. The answer is not as straightforward as it may seem. This article will look at what HEPA filters are made of and examine different types of HEPA filters to better understand if they can be cleaned, and if so, how to do it properly to ensure the filter continues to function according to its claims. HEPA filters, or high-efficiency particulate air filters, are defined by how well they have been rated to filter particles. To meet the HEPA standard, a filter is estimated to remove 99.97 percent of particles that are 0.3 microns (or micrometers) in size from the air that passes through. Originally, HEPA filters were used in research and industrial facilities, because they were developed specifically to remove radioactive particles from the air in nuclear testing labs. Today, HEPA filters show up in a variety of consumer products. A HEPA filter in your HVAC system, air purifier or vacuum cleaner is designed to filter particles from the air in an effort to improve air quality. Studies have indicated that air purifiers with HEPA filters may help reduce the amount of particulates in the air to improve the overall air quality (Laumbach, Meng, & Kipen, 2015). However, some harmful pollutants, like most airborne chemicals, are much smaller than a HEPA filter can trap. As a result Almost all HEPA air purifiers will also have some additional technologies to deal with airborne chemicals. If the filter is marketed as being washable or permanent, then it is possible that you can wash it or clean it off and it will still function. However, there is no standard for washable HEPA filters, and there have not been public studies testing how well these filters work after they have been washed. It is possible that the manufacturer has found a method of making filter fibers that will not be damaged by cleaning, but there really is no way of knowing for sure. The general recommendation is that HEPA filters should be replaced, not cleaned. But if you absolutely have to clean a HEPA filter, there are two ways to do it. Which method you use depends on what type of HEPA filter you have. Because there are no official standards for cleanable HEPA filters, there are no defined terms for the types of cleanable filters available. However, manufacturers have adopted certain marketing terms with some consistency: washable and permanent. A washable HEPA filter should be cleaned by rinsing it under cold water. You should be careful not to touch the filter material, only allowing it to come in contact with water. Allow the filter to completely dry before reinstalling it. Some filters have special instructions. For instance, some canister type vacuum filters should only be washed on the outside of the filter, being careful to avoid getting the central part of the canister wet. It features two large HEPA+Carbon filters that together form a 360-degree cylinder. The HEPA layer captures allergens, bacteria, mold spores, pollen, and other pollutants, while the activated carbon layer removes gases and odors. To comply with the current baseline standard(Opens in a new window), HEPA filters are tested and certified to remove at least 99.97% of dust particles as small as 0.3 microns. Dyson says its filters also meet the HEPA H13 standard(Opens in a new window), meaning they can remove 99.95% of particles down to 0.1 microns. As Wirecutter(Opens in a new window) reported last year, a NASA study found that HEPA filters can actually capture almost 100% of particles as small as 0.01 micron (10 nanometers), well exceeding the baseline standard. To put that in perspective, COVID-19 is about 60 to 140 nanometers(Opens in a new window) in diameter. Dyson doesn't specifically mention COVID-19, but says the TP07 has been scientifically tested to capture airborne viruses, including H1N1 influenza, which is about 80 to 120 nanometers(Opens in a new window) in size. After unboxing the unit, just remove all the protective packing materials, unlock and remove the outer covers, attach the HEPA+Carbon filters to the inside of the cover (you'll hear a clicking sound when they're secure), and put the covers back on the machine. Then, plug it in and use the power button on the unit or the included remote to turn it on. The remote magnetically attaches to the top of the blade, a handy feature since it's small and easy to lose. I then repeated the test using the LG purifier, making sure to put it in the same spot where the TP07 was. Again, the room started out in the green with the PuriCare 360 Single Filter reporting PM1.0, PM2.5, and PM10 levels less than 10. I put the LG purifier in Booster mode with the fan set to Auto (which automatically adjusts the fan speed based on the air quality) and lit the palo santo. It took the LG model two minutes to detect a high level of smoke, then around 20 minutes to purify the air. The machine's filters aren't washable, so you'll need to dispose of them when they're dirty. Dyson says that you should replace the filters about every 12 months, but their lifespan will obviously depend on how often you use the machine. As the filters get clogged with pollutants over time, the machine may begin emitting an unpleasant odor, which is an indication that it's time to change the filters. You can also check your remaining filter life at any time; just press the information button on the remote, or navigate to Settings > Your Machine's Settings in the Dyson Link app. Replacement filters for the Purifier Cool TP07 cost $79.99, which includes both pieces. If you're worried about the cost of filters, or you're looking for a waste-free air purifier for environmental reasons, it's worth checking out the $599 Airdog X5, which uses ionic filtration to rid your air of common pollutants and features washable collecting plates. I prefer the HEPA-based Purifier Cool TP07, which is more compact and doubles as a fan, but the Airdog cuts down on maintenance costs and is a more waste-conscious option. Once these are in place, you replace the outer decorative screens and away you go. According to Dyson, using genuine Dyson filters ensures that your machine continues to work effectively, capturing 99.97% of particles as small as 0.3 microns from your home. The purifier has a dedicated where the machine monitors and purifies using the quietest settings, with a dimmed display. There is also a built-in sleep timer, where you can schedule when your machine turns itself off. 59ce067264 https://www.levelupplumbing.net/forum/general-discussions/buy-finpecia-online-india

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7.3The Big Short 7.3The Big Short > https://urluso.com/2tlJ2o A view of the lateral skull is dominated by the large, rounded cranium above and the upper and lower jaws with their teeth below (Figure 7.3.3). Separating these areas is the bridge of bone called the zygomatic arch. The zygomatic arch (cheekbone) is the bony arch on the side of skull that spans from the area of the cheek to just above the ear canal. It is formed by the junction of two bony processes: a short anterior component, the temporal process of the zygomatic bone and a longer posterior portion, the zygomatic process of the temporal bone, extending forward from the temporal bone. Thus the temporal process (anteriorly) and the zygomatic process (posteriorly) join together, like the two ends of a drawbridge, to form the zygomatic arch. One of the major muscles that pulls the mandible upward during biting and chewing, the masseter, arises from the zygomatic arch. Length is the size of the packet including the 8bytes in the packet header. It is the number of bytes from the start of thisheader to the start of the next packet header. Length is a 2-byte,unsigned short and is represented in network byte order (big-endian). Maternal (intrauterine) influences have been difficult to define but poor maternal nutrition is the most important factor leading to low birth weight and length world-wide (Box 7.13). Maternal alcohol ingestion and smoking are other adverse factors on fetal growth, and maternal infections such as rubella, toxoplasmosis and cytomegalovirus lead to many abnormalities, as well as short stature. Congenital HIV infection also retards fetal growth. Intrauterine growth retardation (IUGR) is usually defined as a birth weight of less that the 10th percentile for gestational age but of these about 10% are not truly abnormal. From the foregoing, it is obvious that GH deficiency and resistance to GH, caused by a genetic defect in the GH receptor, leads to a deficiency in IGF-1 (Box 7.23). Somatotrophin resistance, known as the clinical syndrome of Laron dwarfism, is characterized by high plasma GH and low IGF-1 concentrations and absence of GHBP in most patients. The clinical picture is associated with dysmorphism particularly affecting the central face (prominent forehead and depressed nasal bridge) and marked short stature, the degree being to some extent related to the severity of the functional defect in the receptor. Other features include obesity and delayed puberty. It is rare and treatment with recombinant human IGF-1 markedly improves growth. Prolactin is a single chain protein made up of 199 amino acids with three disulfide bridges and, as noted above, it shares strong structural homologies with GH. Their cell-surface receptors are also similar, although the intracellular domain of the prolactin receptor is different and shorter than that for GH. The intracellular signal transduction pathway is similar to that of GH involving the JAK-STAT kinase pathway (see Box 7.21) and like the GH receptor, truncated forms of the prolactin receptor are found in a number of tissues. Like GH, prolactin secretion is regulated by a dual hypothalamic inhibitory and stimulatory system and can regulate its own secretion through a short-loop feedback (Box 7.15). In contrast, its hypothalamic control is unique in that the predominant hypothalamic influence is inhibitory whereas for all other hormones the predominant influence is stimulatory. Thus, damage to the hypothalamic control causes increased prolactin secretion rather than decreased secretion, as seen with all other anterior pituitary hormones. In general obesity increases with age, and is higher in women and (in Western countries) those from lower socio-economic strata. In many countries, it has reached epidemic proportions and it is more prevalent in certain ethnic groups. The distribution of fat is sexually dimorphic with more subcutaneous fat in women in general and an increase in intra-abdominal fat in men. There is good experi-mental evidence that these two types of adipose tissue behave metabolically differently. The gynecoid distribution leads to low waist-hip circumference ratios whilst android distribution leads to a high ratio. Android obesity is particularly associated with insulin resistance and increased cardiovascular morbidity and mortality (i.e. risk of heart attacks). It is the etiological association with other diseases that makes obesity important (Box 7.36), shortening life-expectancy and reducing its quality. A 22-year-old man was referred to the obesity clinic. With a height of 1.79 m he had weighed 73 kg until 2 years previously. Over the succeeding 2 years his weight had progressively increased to 170 kg and over the same period he had become more withdrawn and had lost his job as a handyman. He had been treated by his primary care physician for depression with little success. There was no family history of obesity. On examination, he looked withdrawn and initiated little spontaneous conversation. In the light of his neurological symptoms, an MR scan of the brain was performed that revealed a large hypothalamic mass with an additional lesion in the brain stem (Box 7.38). The list of possible diagnoses for such multiple lesions includes the granulomatous disease sarcoidosis that affects many organs. Chest X-ray was normal and serum biochemistry showed normal calcium concentration but abnormal tests of liver function. A liver biopsy was, therefore, performed and the typical appearances of sarcoid (non-caseating granulomata) were seen on light microscopy. Psychometric testing showed a major problem with short-term memory. He was treated with oral glucocorticoid steroids with considerable improvement in his mental state and memory. His appetite remained unchanged however, and he lost very little weight despite documented shrinkage of the hypothalamic sarcoid tissue (Box 7.38). Additional endocrine replacement treatment for panhy-popituitarism was also required. \"Lots of seismologists have worked on that problem for many decades. We're not predicting earthquakes in the short term,\" Stanford University geophysics professor Greg Beroza told Vox. \"That requires us to know all kinds of information we don't have.\" In 2003, most of the one-ton SRW 2WD crew cab short-bed trucks offered a 9,900-pound GVWR with a payload rating around 3,200 pounds. Similarly, for a DRW 2WD truck, a one-ton crew cab long-bed truck offered a 11,500-pound GVWR with a payload rating about 5,000 pounds. These were respectable numbers in 2003, but pale in comparison with 2022 Ford F350 numbers, which are a good 1,000 to 2,000 pounds higher. All F350 SRW trucks were equipped with a the aforementioned Dana 60 in the front and a Sterling 10.5 in the rear, while F350 duallys came with a Dana 60 in the front and a Dana 80 rear axle with a GAWR of 11,000 pounds. The Ford F350 came with non limited slip and limited slip rear axles in the following ratios: 3:73, 4:10, 4:30, and 4:56. The 2003 table below is a representative sample the payload ratings of this era. Some minor differences in ratings will be noted between 1995 and 2003 trucks. The easing of inflation pressures helped give workers a lift after months of seeing wage increases fall well short of inflation. Real average hourly earnings rose 0.5% for the month, though they were still down 1.9% from a year ago. Used vehicle prices, which had been a major contributor to the initial inflation burst, fell 2.9% for the month and are now down 3.3% from a year ago. As recently as February, the used cars and trucks index was up more than 40% on an annual basis, the result of higher demand as a microchip shortage caused a backlog in new car production. After spending months dismissing the inflation surge as \"transitory,\" Federal Reserve officials began raising interest rates in March. The central bank has boosted its short-term borrowing rate six times in all, pushing the benchmark up to a targeted range of 3.75%-4%. Regarding isomers, the more branched the chain, the lower the boiling point tends to be. London dispersion forces are smaller for shorter molecules and only operate over very short distances between one molecule and its neighbors. It is more difficult for short, bulky molecules (with substantial amounts of branching) to lie close together (compact) compared with long, thin molecules. Cycloalkanes are similar to alkanes in their general physical properties, but they have higher boiling points, melting points, and densities than alkanes. This is due to stronger London forces because the ring shape allows for a larger area of contact. When a bond is strong, there is a higher bond energy because it takes more energy to break a strong bond. This correlates with bond order and bond length. When the Bond order is higher, bond length is shorter, and the shorter the bond length means a greater the Bond Energy because of increased electric attraction. In general, the shorter the bond length, the greater the bond energy. Between 1930 and 1933, Steinbeck produced three shorter works. The Pastures of Heaven, published in 1932, consists of twelve interconnected stories about a valley near Monterey, which was discovered by a Spanish corporal while chasing runaway Indian slaves. In 1933 Steinbeck published The Red Pony, a 100-page, four-chapter story weaving in memories of Steinbeck's childhood.[21] To a God Unknown, named after a Vedic hymn,[16] follows the life of a homesteader and his family in California, depicting a character with a primal and pagan worship of the land he works. Although he had not achieved the status of a well-known writer, he never doubted that he would achieve greatness.[16] In 1952, John Steinbeck appeared as the on-screen narrator of 20th Century Fox's film, O. Henry's Full House. Although Steinbeck later admitted he was uncomfortable before the camera, he provided interesting introductions to several filmed adaptations of short stories by the legendary writer O. Henry. About the same time, Steinbeck recorded readings of several of his short stories for Columbia Records; the recordings provide a record of Steinbeck's deep, resonant voice. 59ce067264 https://www.shul.org.au/group/mysite-231-group/discussion/623da668-46d3-4596-80ee-1e526734560b

Arctic Archipelago DOWNLOAD >> https://urlgoal.com/2tlAWG The Arctic Archipelago, also known as the Canadian Arctic Archipelago, is an archipelago lying to the north of the Canadian continental mainland, excluding Greenland (an autonomous territory of Denmark) and Iceland (an independent country). Situated in the northern extremity of North America and covering about 1,424,500 km2 (550,000 sq mi), this group of 36,563 islands, surrounded by the Arctic Ocean, comprises much of Northern Canada, predominately Nunavut and the Northwest Territories.[5] The archipelago is showing some effects of climate change,[6][7] with some computer estimates determining that melting there will contribute 3.5 cm (1.4 in) to the rise in sea levels by 2100.[8] The archipelago extends some 2,400 km (1,500 mi) longitudinally and 1,900 km (1,200 mi) from the mainland to Cape Columbia, the northernmost point on Ellesmere Island. It is bounded on the west by the Beaufort Sea; on the northwest by the Arctic Ocean; on the east by Greenland, Baffin Bay and Davis Strait; and on the south by Hudson Bay and the Canadian mainland. The various islands are separated from each other and the continental mainland by a series of waterways collectively known as the Northwest Passage. Two large peninsulas, Boothia and Melville, extend northward from the mainland. The northernmost cluster of islands, including Ellesmere Island, is known as the Queen Elizabeth Islands and was formerly the Parry Islands. The archipelago consists of 36,563 islands, of which 94 are classified as major islands, being larger than 130 km2 (50 sq mi), and cover a total area of 1,400,000 km2 (540,000 sq mi).[14] The islands of the archipelago over 10,000 km2 (3,900 sq mi), in order of descending area, are: After Greenland, the archipelago is the world's largest high-Arctic land area. The climate of the islands is Arctic, and the terrain consists of tundra except in mountainous regions. Most of the islands are uninhabited; human settlement is extremely thin and scattered, being mainly coastal Inuit settlements on the southern islands. The major islands in the eastern Arctic (Baffin, Devon, Ellesmere,Axel Heiberg) are mountainous, with peaks over 2,000 m. The higher land on these islands is commonly occupied by ice caps that contain most (75 per cent) of the glacierice and the largest glaciers in Canada, and one-third of the volume of land ice worldwide, not including the ice sheets found on Greenland and Antarctica. These highland areas were the major source area for the Innuitian ice sheet during the last glaciation(see also Innuitian Region). The sea ice cover, with an average thickness of about 1.5 to 2 m is complete in winter throughout the archipelago, with the exception of several recurring polynyas (areas of open water surrounded by sea ice), the largest of which is in northern BaffinBay and in the southeastern Beaufort Sea. These polynyas, which freeze late and thaw early, are a focus for marine and bird life. There is evidence of early human habitation,by the early Inuit (Thule) and Dorset peoples, on the landmasses adjoining them. As in the case of plants, the number of animal species decreases north from the mainland. Some 20 species of land mammals live on the archipelago, generally in small numbers restricted to certain areas. The High Arctic islands are home to Peary caribouthat are smaller and lighter in colour than the barren ground caribou. Other mammals in the archipelago include the muskox, Arctic fox, Arctic wolf, lemmingand Arctic hare. More than 60 species of birds spend the summer in the High Arctic islands, while only six species overwinter there. The surrounding seas are home to thepolar bear, the walrus and various types ofseal and whale, including thenarwhal and the beluga. During the 1950s, Canadian government, university and private sector research and exploration expanded. Aerial photo coverage of the archipelago was completed and the Geological Survey of Canada was very active, mapping the geology and determining, amongother things, the presence of oil in the islands. McGill University expeditions to Axel Heiberg Island and the Arctic Institute of North America (now University ofCalgary) expeditions to Devon Island began regular glacier measurements that continue to this day as a way of monitoring glacier change in the region. Today, Resolute, CambridgeBay and Nanisivik (Northern Baffin Island) are developing centres of research and military activity. Though much attention has been focused in recent years on the melting of ice from Greenland and Antarctica, nearly half of the ice volume currently being lost to the ocean is actually coming from other mountain glaciers and ice caps. Ice loss from a group of islands in northern Canada accounts for much of that volume. The Arctic is warming more rapidly than other region on the planet, and the northern Barents Sea, including the Svalbard Archipelago, is experiencing the fastest temperature increases within the circumpolar Arctic, along with the highest rate of sea ice loss. These physical changes are affecting a broad array of resident Arctic organisms as well as some migrants that occupy the region seasonally. Herein, evidence of climate change impacts on terrestrial and marine wildlife in Svalbard is reviewed, with a focus on bird and mammal species. In the terrestrial ecosystem, increased winter air temperatures and concomitant increases in the frequency of 'rain-on-snow' events are one of the most important facets of climate change with respect to impacts on flora and fauna. Winter rain creates ice that blocks access to food for herbivores and synchronizes the population dynamics of the herbivore-predator guild. In the marine ecosystem, increases in sea temperature and reductions in sea ice are influencing the entire food web. These changes are affecting the foraging and breeding ecology of most marine birds and mammals and are associated with an increase in abundance of several temperate fish, seabird and marine mammal species. Our review indicates that even though a few species are benefiting from a warming climate, most Arctic endemic species in Svalbard are experiencing negative consequences induced by the warming environment. Our review emphasizes the tight relationships between the marine and terrestrial ecosystems in this High Arctic archipelago. Detecting changes in trophic relationships within and between these ecosystems requires long-term (multidecadal) demographic, population- and ecosystem-based monitoring, the results of which are necessary to set appropriate conservation priorities in relation to climate warming. Seabirds are thought to provide ecological services such as the movement of nutrients between marine and terrestrial ecosystems, which may be especially critical to productivity and diversity in nutrient-poor environments. Most Arctic ecosystems are unaffected by local human impacts and are naturally nutrient poor and especially sensitive to warming. Here, we assessed the effects of nesting common eider ducks (Somateria mollissima) on soil, vegetation, and pond sediments on island archipelagoes in Hudson Strait between Nunavut and Québec, Canada. Soil, moss, and pond sediments were significantly higher in nitrogen on islands with large numbers of nesting eiders compared to sites with no nesting birds. The highest concentrations of nitrogen in soils and moss occurred at the margins of ponds on eider islands, which correspond to the areas of highest eider use. δ15N and δ34S values in soils, moss, and sediments indicated substantial marine-derived organic matter inputs at the higher nutrient sites. We propose that by foraging on coastal marine benthic invertebrates and returning to islands to nest, eider ducks bio-transport and concentrate marine-derived nutrients to their colony islands, fertilizing Arctic island ecosystems in the process. As common eiders nest on thousands of low to mid-latitude islands throughout the circumpolar Arctic, these nutrient inputs likely dramatically affect biota and ecosystem functioning throughout the tundra biome. The islands of the Hudson Strait region present an important study opportunity as these islands are representative of thousands of similar low-lying, nearshore coastal islands across a large area of the eastern Canadian Arctic and West Greenland, many of which are used for nesting by northern common eiders (Somateria mollisima borealis). Whereas other studies have demonstrated seabirds that breed in very large colonies or at very high densities, and that feed at high trophic levels (e.g. are piscivorous), can be effective bio-vectors of nutrients (e.g., Keatley et al. 2011; Caut et al. 2012; Hargan et al. 2017), few studies have investigated whether seabirds that feed at lower trophic levels (e.g., feed on invertebrates) supplement nutrient levels in the terrestrial environment at or near their colonies. However, one study from the Canadian High Arctic did demonstrate that eiders deliver distinct contaminant mixtures to ponds that is distinguishable from a pond predominantly influence by nesting arctic terns (Michelutti et al. 2010). Still fewer studies have investigated whether important nutrient inputs occur across large geographical scales, such as large island archipelagos (but see Maron et al. 2006). Our results, taken together, strongly suggest that common eider ducks are acting as ecologically important bio-vectors of marine nutrients to the terrestrial environment of the Arctic islands where they nest, both in small numbers and large. As common eider ducks nest throughout Hudson Strait, including Ungava Bay and the surrounding areas (Cooch 1986; Iverson et al. 2014), our study demonstrates that these marine-derived nutrients could have broad regional-scale effects on soil and water chemistry across these island archipelagos. Abstract. In the Arctic Ocean region, methane concentrations are higher than the global average; high concentrations of dissolved CH4 are detectable especially across many subarctic and Arctic continental shelf margins. Yet the Arctic Ocean appears to emit only minimal methane fluxes to the atmosphere across the air-sea interface, suggesting water column oxidation of methane may be an important process. Here we paired thermohaline, chemical, and biological data collected during the Northwest Passage Project transit through the Canadian Arctic Archipelago (CAA) waters in the Summer of 2019, with in-situ and in-vitro methane data. Our results showed high meltwater (meteoric water + sea ice melt) throughout the Western CAA and Croker Bay in the East, and these surface meltwaters showed methane excess. The meteoric waters showed a strong correlation with chlorophyll-α fluorescence (r=0.63), as well as a correlation between dissolved [CH4] and chlorophyll-α fluorescence (r=0.74). Methane oxidation rate constants were highest in Wellington Channel and Croker Bay surface waters (av. 0.010 d-1), characterized by meltwaters and Pacific-origin waters. The average oxidation rates in meteoric and Pacific waters were respectively 24.4 % and 12.6 % higher than the entire survey average. Moreover, Pacific and meteoric waters hosted microbial taxa of Pacific-origin that are associated with methane oxidation, Oleispira (γ-proteobacteria), and Aurantivirga (Flavobacteria). The deeper layers were characterized by low methane concentrations and low methane oxidation rate constants (av. 0.0040.002 d-1). Sea ice covered much of the Western CAA, in the same region with high sea ice meltwater concentrations. These waters also hosted higher average methane oxidation rates (av. 0.0070.002 d-1). To the east, open coastal water coincided with methane enrichment, but low chlorophyll fluorescence and weak methane oxidation. These results suggest that methane production in ice-associated Arctic blooms may be quickly oxidized by microbes that are also found in these waters, associated with seasonal biology. 59ce067264 https://www.seashellsandkettlebells.com/group/5-weeks-to-5k-group-discussion/discussion/54b71d26-22cb-4f91-8fa8-d7625146e328

Buying The Bayou Episode List Buying The Bayou Episode List >> https://bytlly.com/2tlA5d Porter 2:36 Well, thanks a lot for coming on. We found a few Gremlins while getting you here. So let's hope everything holds for the rest of the podcast. So, we'll have a very exciting episode today with the both of you on buying and selling races - awesomely interesting topic - that lots of people would want to know more about. But before we get into all that, let's go over some introductions, shall we So please introduce yourselves - where are you based, your company, the races you put on and, basically, what do you do within the industry. Panos 4:23 Okay, great. So as I said, today, we'll talk about buying and selling races. It's a topic that I think most race directors - at least, the commercial for-profit companies - have thought about. They must have thought about some kind of exit for the business or a way to expand their business by buying more races. So I think that will be a really interesting topic for them. And I chose the two of you because you've both sold and bought races. I think Porter told me that he had a lot of experience buying some races - how he reached out to the right people and, like, that whole process. And then, Tony, of course, you sold your series of races. You went through the business brokers, all that stuff, which is also going to be very interesting for people. So in brief - maybe Tony can start first - tell our listeners a little bit about your background in buying and selling events. Tony 12:37 Yeah, so we talked about that a little bit. The issue was the relationship between some of the other race directors in the area and we didn't feel comfortable in advertising that, \"Hey, we're looking at getting out of the space in the Houston area.\" And just the sheer size of our events-- I mean, like I mentioned, we were pulling almost 20,000 people a year for the events that we were looking to offload. When we made the decision to sell, it wasn't a requirement that we sell the races because we're still doing good. They were pulling in numbers. The margins were hanging right around where they should be for an event and they were bringing positive cash flow to the business. We didn't have to sell. When we did so, it wasn't something where we go out and advertise with a big \"For Sale\" sign or put up a billboard in the city or anything. I think that, kind of, drove how we ended up listing through a broker versus us trying to sell it ourselves. It kind of separated us from the-- like what Porter mentioned, \"This is our baby. We grew it from the ground up and we built it to what it is. So it's worth millions upon millions of dollars because of all the hard work we put in to do that.\" It's just not the case whenever we try to look at what the actual worth of an event is and how you calculate the kind of return you get on the buying and selling side. My buying experience is more towards buying a percentage of ownership of events versus full events - so not quite as extensive as Porter. I just wanted to try and separate that emotional attachment that we have in the process and how we come up with the numbers and everything. Tony 14:46 So we reached out to three different brokers that have a pretty diversified portfolio of selling different kinds of businesses. There are brokers out there that specialize in construction and things of that nature. Because we knew that there's not a lot of races going up for sale, we wanted somebody that had, kind of, a diverse portfolio of different businesses that they sold - so that was kind of our first criteria. Once we got beyond that, obviously, we needed communication - I wanted to make sure they got back to me. So if I reached out to somebody and they didn't email me back for a week, that was an immediate red flag because this is going to be a process and we knew it was going to be. And then, of course, the fees. At the end of the day, it all comes down to how much money you put in your pocket when you try to sell an event. So it was kind of a balancing act. And the one we ended up settling on wasn't the cheapest, but it was the one we felt most comfortable with at the time and they were great. Basically, the process was they provide a worksheet - it was about 10 pages long - with questions. Some were really simple questions. Some were a little more complicated where we had to do a little more digging. And that was the first step. And they built our, basically, public-facing prospectus out of that worksheet. And it was just a listing on their website that they had and they have their own other marketing tools for potential buyers. And then, after that, it was just paperwork, lots of different requests - books, financial reports, any outstanding bills, balances, things like that - and just keeping that information up there. We were on the market for a good nine months before we got the person that ultimately ended up buying the races. So, it was definitely a process of keeping things updated so that all these information are available whenever somebody did approach them. Porter 29:42 Yeah, it's an asset purchase allocation - that's what it's called. There are different classes of assets whenever a business is formed. And in our purchase agreement, we actually have a table listing of the allocation. Like, Class 1 is cash and cash equivalents that you have on hand. Class 2 is any trade and investments. Accounts receivable is Class 3. Consumables is Class 4. So we had some of those because that was Gatorade and cups and things like that. Tangible fix - that would be the equipment. Noncompetition agreements - you do apply a value to non-compete that you implement whenever you're buying and selling. And then, any intangible assets - that'd be all your URLs and your social media. And, then, class seven, the final one is goodwill. And basically, usually, at least, what happened in our case was we worked with the buyer and divided out the purchase price, and said, \"How much are the consumables worth\" \"Well, it's this much.\" And we, kind of, broke it out that way. And then, everything else is goodwill. Tony 37:16 You know, of the many things that you'll need to worry about when selling your race, keeping a clean record of your event's data, being able to share that data with buyers and, when it comes to pulling the trigger, being able to transfer that data on to the next race director is right there at the top of the list. And I can tell you there's no better platform to do all that on than GiveSignupRunSignup. With GiveSignupRunSignup, it's easy to shift backend race access from one race director to another, providing third parties of your choosing with instant insights into historical participant data and communications. In fact, every single historical action a participant may have taken on your race, from using a coupon code to making a donation, is stored right there on your race director dashboard. What does that mean Well, it means that you can boast to the buyer being able to transfer over not just a list of emails, but a full CRM system that your event's buyer can then use to extract more value from your race - meaning, an easier and more profitable transaction for you, the seller. On top of that, you can make use of GiveSignupRunSignup's extensive reporting functionality to quickly furnish potential buyers with all kinds of valuable insights from historical registration patterns to repeat participant data - both super important metrics that can demonstrate the quality of your annual registration revenue. And, of course, these are all tools you can benefit from for many years before you decide to sell your events. Keeping your data in good order is just something you'd want to do. And when the time comes to transition your events, it will make your life a whole lot easier. So, to learn more about GiveSignupRunSignup's amazing technology platform, head over to run sign up.com That's runsignup.com. And while you're there - because there's so many great features to go over - my advice would be to just hit the \"Schedule a demo\" button and let one of the team walk you through how everything comes together. You will not regret it. In the meantime, we still have plenty of these very interesting episode ahead of us, including valuing your event and negotiating the sale. So, let's get back to buying and selling races with Tony Sapp and Porter Bratten. Porter 51:37 I think it's mostly because of the different industry and the status of the business. A lot of the Shark Tank businesses - they're either software or they're startups and things like that. So they don't really have a history of revenue to go off of. What I think a lot of the sharks or dragons are buying in those shows are the potential, the future, what they could grow it to, or use it as part of their business portfolio. I mean, you may be able to extract something more. If you went to a high-level capital investor or something like that, I mean, we see some pretty big multiples get thrown around with the Eventbrite IPO and things like that. None of us are Iron Man. We're not huge organizations with lots of people or with hundreds of employees around the world. The people that are listening to this podcast are like Porter and I - we're 1-4 person businesses. Maybe, there are a few part-timers on top of that. It's not a corporation that's offing assets. It's a small local company for the most part. 59ce067264 https://www.unifiedbjj.com/group/novice-karate-group-ages-8-up/discussion/6f9f76ab-f42d-4e24-8541-a1aaff7fcaa0

Buy Quadro P4000 CLICK HERE >>> https://bltlly.com/2tlz9u So I'm going to build a new PC for rendering in Daz, I heard a lot of people talkin about the quadro graphic cards, but I saw that quadro p4000 and GTX 1080 TI have almost the same price but Quadro have half of the cuda cores that GTX has. For daz IRay. What do you guys recomand between a quadro and a GTX? No entries for the p4000, but I doubt it would beat a p5000, which benches only marginally better than a 1070, so James' estimation could be pretty accurate (but I think it is more likely way too over optimistic). The p4000 isn't a particularly impressive card. Quadros until recently weren't even a consideration for GPU rendering. They are much better now, but in terms of raw performance you will still get better performance for cost from consumer cards. Consumer cards tend to have better cooling too. Do you realize that the Pxxxx series quadro cards are running the same Pascal chips that are on the GTX 10xx cards?Do you also realize that you can get Pascal based Quadro cards with much larger amounts of Vram?The two biggest differences between Quadro cards and Geforce cards: P series Quadros do not turbo boost, and the drivers are geared towards CG work instead of gaming. If *all* that is going to be done is Iray under DS on a consumer grade desktop then using a Quadro is not likely to be the best dollar-for-dollar. If, however, you are doing more than simple scene renders then a quadro is the better solution. Here is the eye watering Quadro P6000 24Gb -nvidia-quadro-p6000-24gb-workstation-graphics-card this is Australian pricing but as you can see you could by an uber up to date complete system for what this card costs.. But would be very nice all the same.. 59ce067264 https://www.benchwalklaw.com/group/benchwalklaw-com-group/discussion/06ede0ce-0d85-4301-ab2a-1e9c232fef54 https://www.girl-alliance.com/forum/untitled-category/animal-shelter-free-download